Glucose availability regulates nicotinamide N-methyltransferase expression in adipocytes

Author:

Ehebauer Franziska,Ghavampour Sharang,Kraus Daniel

Funder

Deutsche Forschungsgemeinschaft

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference28 articles.

1. Nicotinamide N-methyltransferase knockdown protects against diet-induced obesity;Kraus;Nature,2014

2. Selective and membrane-permeable small molecule inhibitors of nicotinamide N-methyltransferase reverse high fat diet-induced obesity in mice;Neelakantan;Biochem. Pharmacol.,2017

3. Adipose tissue as a source of nicotinamide N-methyltransferase and homocysteine;Riederer;Atherosclerosis,2009

4. Glucose transporters and insulin action--implications for insulin resistance and diabetes mellitus;Shepherd;N. Engl. J. Med.,1999

5. Tissue-specific ablation of the GLUT4 glucose transporter or the insulin receptor challenges assumptions about insulin action and glucose homeostasis;Minokoshi;J. Biol. Chem.,2003

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