JAK/STAT signaling is involved in IL-35-induced inhibition of hepatitis B virus antigen-specific cytotoxic T cell exhaustion in chronic hepatitis B-Reference-Cited by-同舟云学术

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JAK/STAT signaling is involved in IL-35-induced inhibition of hepatitis B virus antigen-specific cytotoxic T cell exhaustion in chronic hepatitis B

Published:2020-07 Issue: Volume:252 Page:117663
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Dong Yuejiao,Li Xuefen,Yu Yanying,Lv Feifei,Chen Yu

Funder

National Natural Science Foundation of China

Major National S&T Projects for Infectious Diseases

Natural Science Foundation of Zhejiang Province

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference36 articles.

1. The global burden of liver disease: the major impact of China;Wang;Hepatology,2014

2. Global epidemiology of hepatitis B virus infection: new estimates of age-specific HBsAg seroprevalence and endemicity;Ott;Vaccine,2012

3. Chemokines enhance immunity by guiding naive CD8+ T cells to sites of CD4+ T cell–dendritic cell interaction;Castellino;Nature,2006

4. Correlation of antiviral T-cell responses with suppression of viral rebound in chronic hepatitis B carriers: a proof-of-concept study;Yang;Gene Ther.,2006

5. Immunology of hepatitis B virus and hepatitis C virus infection;Rehermann;Nat. Rev. Immunol.,2005

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4. Decreased Expression of IL‐35 and Its Receptor Contributes to Impaired Megakaryopoiesis in the Pathogenesis of Immune Thrombocytopenia;Advanced Science;2024-01-15

5. Evaluation serum level of IL-17, IL-23 and IL-35 in patients with bacterial prostatitis in Najaf Province;AIP Conference Proceedings;2024

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