ApoE deficiency promotes non-alcoholic fatty liver disease in mice via impeding AMPK/mTOR mediated autophagy-Reference-Cited by-同舟云学术

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ApoE deficiency promotes non-alcoholic fatty liver disease in mice via impeding AMPK/mTOR mediated autophagy

Published:2020-07 Issue: Volume:252 Page:117601
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Lu Wanpeng,Mei Jinyu,Yang Juan,Wu Zhihan,Liu Jiayuan,Miao Pengyu,Chen Yiliang,Wen Zhenfan,Zhao Zhongting,Kong Hua,Wu Chao,Yang Yan,Chen Ming

Funder

National Natural Science Foundation of China

Anhui Medical University

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference51 articles.

1. En nombre del grupo de trabajo sobre de la Societat Catalana de D. Non-alcoholic fatty liver disease: a poorly known pandemic;Augustin;Med. Clin. (Barc.),2017

2. Non-alcoholic fatty liver disease;Neuschwander-Tetri;BMC Med.,2017

3. From fatty liver to fibrosis: a tale of “second hit”;Basaranoglu;World J. Gastroenterol.,2013

4. Puerarin protects against high-fat high-sucrose diet-induced non-alcoholic fatty liver disease by modulating PARP-1/PI3K/AKT signaling pathway and facilitating mitochondrial homeostasis;Wang;Phytother. Res.,2019

5. Nonalcoholic fatty liver disease: pathogenesis and therapeutics from a mitochondria-centric perspective;Gusdon;Oxidative Med. Cell. Longev.,2014

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