VHL gene methylation contributes to excessive erythrocytosis in chronic mountain sickness rat model by upregulating the HIF-2α/EPO pathway-Reference-Cited by-同舟云学术

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VHL gene methylation contributes to excessive erythrocytosis in chronic mountain sickness rat model by upregulating the HIF-2α/EPO pathway

Published:2021-02 Issue: Volume:266 Page:118873
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Yang Min,Zhu Mingming,Song Kang,Wuren Tanna,Yan Jun,Ge Ri-Li,Ji Linhua,Cui Sen

Funder

Qinghai University

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference50 articles.

1. Consensus statement on chronic and subacute high altitude diseases;León-Velarde;High Alt. Med. Biol.,2005

2. D. Maimaitiyimin, A. Aikemu, M. Kamilijiang, A. Salamu, X. Zhang, Effects and mechanisms of acetyl-L-cysteine in rats with chronic mountain sickness with H1-NMR metabolomics methods, Med. Sci. Monit. 20 (2014) 767–773. doi:10.12659/MSM.890244.

3. High-altitude disorders: pulmonary hypertension: pulmonary vascular disease: the global perspective;Pasha;Chest.,2010

4. Chronic mountain sickness: clinical aspects, etiology, management, and treatment;Villafuerte;High Alt. Med. Biol.,2016

5. The local HIF-2alpha/EPO pathway in the bone marrow is associated with excessive erythrocytosis and the increase in bone marrow microvessel density in chronic mountain sickness;Su;High Alt. Med. Biol.,2015

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4. DNA methyltransferase 3A induces the occurrence of oral submucous fibrosis by promoting the methylation of the von Hippel–Lindau;Oral Diseases;2023-09-24

5. New characteristics in ECG induced by ultra-high altitude hypoxia;Biomedical Signal Processing and Control;2023-08

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