Calcium-independent CaMKII activity is involved in ginsenoside Rb1-mediated neuronal recovery after hypoxic damage-Reference-Cited by-同舟云学术

Calcium-independent CaMKII activity is involved in ginsenoside Rb1-mediated neuronal recovery after hypoxic damage

Published:2005-01 Issue:9 Volume:76 Page:1013-1025
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Park Jin Kyu,Namgung Uk,Lee Chang Joong,Park Jong Oh,Jin Sung-Ha,Kwon Oh-Bin,Ko Sung Ryong,Kim Sang Won,Kang Eun Jung,Ko Ji Hun,Lee Sang Myung,Kim Dong Hee,Won Moo Ho

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference33 articles.

1. Ischemia-induced translocation of Ca2+/calmodulin-dependent protein kinase II: potential role in neuronal damage;Aronowski;Journal of Neurochemistry,1992

2. Ginseng pharmacology: multiple constituents and multiple actions;Attele;Biochemical Pharmacology,1999

3. Culturing Nerve Cells;Banker,1998

4. Effects of ginsenoside Rb1 on central cholinergic metabolism;Benishin;Pharmacology,1991

5. Quantitative determination of glutamate mediated cortical neuronal injury in cell culture by lactate dehydrogenase efflux assay;Choi;Journal of Neuroscience Methods,1987

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