Disturbance of redox homeostasis by ornithine and homocitrulline in rat cerebellum: A possible mechanism of cerebellar dysfunction in HHH syndrome-Reference-Cited by-同舟云学术

Disturbance of redox homeostasis by ornithine and homocitrulline in rat cerebellum: A possible mechanism of cerebellar dysfunction in HHH syndrome

Published:2013-08 Issue:4 Volume:93 Page:161-168
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Zanatta Ângela,Viegas Carolina Maso,Tonin Anelise Miotti,Busanello Estela Natacha Brandt,Grings Mateus,Moura Alana Pimentel,Leipnitz Guilhian,Wajner Moacir

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico (CNPq)

Programa de Apoio a Núcleos de Excelência (PRONEX II)

Fundação de Amparo à Pesquisa do Estado do Rio Grande do Sul (FAPERGS)

Pró-Reitoria de Pesquisa/Universidade Federal do Rio Grande do Sul (PROPESQ/UFRGS)

Financiadora de estudos e projetos (FINEP)

Rede Instituto Brasileiro de Neurociência (IBN-Net)

Instituto Nacional de Ciência e Tecnologia em Excitotoxicidade e Neuroproteção (INCT-EN)

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference53 articles.

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2. Evidence that the major metabolites accumulating in hyperornithinemia-hyperammonemia-homocitrullinuria syndrome induce oxidative stress in brain of young rats;Amaral;Int J Dev Neurosci,2009

3. Current concepts in the pathogenesis of urea cycle disorders;Braissant;Mol Genet Metab,2010

4. Reduced glutathione and glutathione disulfide;Browne;Methods Mol. Biol.,1998

5. Hyperornithinaemia-hyperammonaemia-homocitrullinuria syndrome is caused by mutations in a gene encoding a mitochondrial ornithine transporter;Camacho;Nat Genet,1999

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