Disruption of dopamine D1/D2 receptor complex is involved in the function of haloperidol in cardiac H9c2 cells

Author:

Lencesova L.,Szadvari I.,Babula P.,Kubickova J.,Chovancova B.,Lopusna K.,Rezuchova I.,Novakova Z.,Krizanova O.,Novakova M.

Funder

VEGA

APVV

Grant Agency of Czech Republic

National Program of Sustainability II

European Regional Development Fund ITMS

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference36 articles.

1. Recognition and management of withdrawal delirium (delirium tremens);Schuckit;N. Engl. J. Med.,2014

2. Typical and atypical antipsychotics do not differ markedly in their reversibility of antagonism of the dopamine D2 receptor;Sahlholm;Int. J. Neuropsychopharmacol.,2014

3. Modulation of expression of the sigma receptors in the heart of rat and mouse in normal and pathological conditions;Novakova;Physiol. Biophys.,2007

4. Haloperidol increases expression of the inositol 1,4,5-trisphosphate receptors in rat cardiac atria, but not in ventricles;Novakova;Gen. Physiol. Biophys.,2010

5. Long-term haloperidol treatment prolongs QT interval and increases expression of sigma 1 and IP3 receptors in guinea pig hearts;Stracina;Tohoku J. Exp. Med.,2015

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