Oxidative damage of rat liver mitochondria during exposure to t-butyl hydroperoxide. Role of Ca2+ ions in oxidative processes-Reference-Cited by-同舟云学术

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Oxidative damage of rat liver mitochondria during exposure to t-butyl hydroperoxide. Role of Ca2+ ions in oxidative processes

Published:2013-06 Issue:23 Volume:92 Page:1110-1117
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Zavodnik Ilya B.,Dremza Iosif K.,Cheshchevik Vitali T.,Lapshina Elena A.,Zamaraewa Maria

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference45 articles.

1. Kinetic model for Ca2+-induced permeability transition in energized liver mitochondria discriminates between inhibitor mechanisms;Baranov;J Biol Chem,2008

2. Agmatine prevents the Ca(2+)-dependent induction of permeability transition in rat brain mitochondria;Battaglia;Amino Acids,2010

3. Heterolytic reduction of fatty acid hydroperoxides by cytochrome c/cardiolipin complexes: antioxidant function in mitochondria;Belikova;J Am Chem Soc,2009

4. Regulation of intracellular calcium compartmentation: studies with isolated hepatocytes and t-butyl hydroperoxide;Bellomo;Proc Natl Acad Sci U S A,1982

5. Pyridine-nucleotide oxidation, Ca2+ cycling and membrane damage during tert-butyl hydroperoxide metabolism by rat-liver mitochondria;Bellomo;Eur J Biochem,1984

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