Role of ion channels in chronic intermittent hypoxia-induced atrial remodeling in rats-Reference-Cited by-同舟云学术

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Role of ion channels in chronic intermittent hypoxia-induced atrial remodeling in rats

Published:2020-08 Issue: Volume:254 Page:117797
ISSN:0024-3205
Container-title:Life Sciences
language:en
Short-container-title:Life Sciences

Author:

Zhang Kai,Ma Zuowang,Song Chen,Duan Xiaorui,Yang Yu,Li Guangping

Funder

National Natural Science Foundation of China

Key Laboratory Science Foundation of Second Hospital of Tianjin

Tianjin Natural Science Foundation

Publisher

Elsevier BV

Subject

General Pharmacology, Toxicology and Pharmaceutics,General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference33 articles.

1. Pathophysiological mechanisms of atrial fibrillation: a translational appraisal;Schotten;Physiol. Rev.,2011

2. Genetic predisposition, clinical risk factor burden, and lifetime risk of atrial fibrillation;Weng;Circulation,2018

3. Computational modeling of electrophysiology and pharmacotherapy of atrial fibrillation: recent advances and future challenges;Vagos;Front. Physiol.,2018

4. Mechanisms of cardiac dysfunction in obstructive sleep apnea;Baguet;Nat. Rev. Cardiol.,2012

5. Estimation of the global prevalence and burden of obstructive sleep apnoea: a literature-based analysis;Benjafield;Lancet Respir. Med.,2019

Cited by 8 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Metabolic remodelling in atrial fibrillation: manifestations, mechanisms and clinical implications;Nature Reviews Cardiology;2024-05-30

2. Chronic intermittent hypoxia‐induced cardiovascular and renal dysfunction: from adaptation to maladaptation;The Journal of Physiology;2023-10-26

3. Mechanisms associated with the development of atrial fibrillation after sepsis and the role of neuregulin-1;2023-08-18

4. Effects of doxycycline on atrial electrical remodeling in a rat model of atrial fibrillation;2023-07-03

5. Mechanisms of Atrial Fibrillation in Obstructive Sleep Apnoea;Cells;2023-06-19

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