Moderate prenatal alcohol exposure increases total length of L1-expressing axons in E15.5 mice-Reference-Cited by-同舟云学术

Moderate prenatal alcohol exposure increases total length of L1-expressing axons in E15.5 mice

Published:2021-05 Issue: Volume:85 Page:106962
ISSN:0892-0362
Container-title:Neurotoxicology and Teratology
language:en
Short-container-title:Neurotoxicology and Teratology

Author:

Sicher Avery,Kiss Shannon,Springmann Paige,Herrera Karen,McElroy Abigail,Blake Kelsey,Crocker Emily,Jacob Christa,Lefkove McKayla,Cramer Myla,Henriksen Allysen,Novacek Josef,Severa Jenna,Siberski Justin,Thomas Emily,Chi Peter,Favero Carlita

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Developmental Neuroscience,Toxicology

Reference45 articles.

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2. Slit proteins prevent midline crossing and determine the dorsoventral position of major axonal pathways in the mammalian forebrain;Bagri;Neuron,2002

3. L1 cell adhesion molecule signal cascades: targets for ethanol developmental neurotoxicity;Bearer;Neurotoxicology,2001

4. Slit2 activity in the migration of guidepost neurons shapes thalamic projections during development and evolution;Bielle;Neuron,2011

5. Sensory processing and adaptive behavior deficits of children across the fetal alcohol spectrum disorder continuum;Carr;Alcohol. Clin. Exp. Res.,2010

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1. Prenatal alcohol exposure alters axon formation and Robo/Slit guidance cue expression in the internal capsule at E15.5;2022-05-12