Cleft palate by picrotoxin or 3-MP and palatal shelf elevation in GABA-deficient mice
Author:
Publisher
Elsevier BV
Subject
Cellular and Molecular Neuroscience,Developmental Neuroscience,Toxicology
Reference28 articles.
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2. Cleft palate and decreased brain γ-aminobutyric acid in mice lacking the 67-kDa isoform of glutamic acid decarboxylase;Asada;Proc. Natl. Acad. Sci. U. S. A.,1997
3. Cleft palate in mice with a targeted mutation in the γ-aminobutyric acid-producing enzyme glutamic acid decarboxylase 67;Condie;Proc. Natl. Acad. Sci. U. S. A.,1997
4. Concordance between isolated cleft palate in mice and alterations within a region including the gene encoding the β3 subunit of the type A γ-aminobutyric acid receptor;Culiat;Proc. Natl. Acad. Sci. U. S. A.,1993
5. Deficiency of the β3 subunit of the type A γ-aminobutyric acid receptor causes cleft palate in mice;Culiat;Nat. Genet.,1995
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