Role of metabolic activation, covalent binding, and glutathione depletion in pulmonary toxicity produced by an impurity of malathion-Reference-Cited by-同舟云学术

Role of metabolic activation, covalent binding, and glutathione depletion in pulmonary toxicity produced by an impurity of malathion

Published:1984-03 Issue:3 Volume:72 Page:476-483
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

Imamura T.,Hasegawa L.

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference19 articles.

1. In vivo studies on the relationship between target organ alkylation and the pulmonary toxicity of a chemically reactive metabolite of 4-ipomeanol;Boyd;J. Pharmacol. Exp. Ther.,1978

2. Protective role of endogenous pulmonary glutathione and other sulfhydryl compounds against lung damage by alkylating agents. Investigations with 4-ipomeanol in the rat;Boyd;Biochem. Pharmacol.,1982

3. Metabolic activation of toxins in extrahepatic target organs and target cells;Boyd,1980

4. Tissue sulfhydryl groups;Ellman;Arch. Biochem. Biophys.,1959

5. Phenobarbital pretreatment protects against morphologic changes in rat bronchiolar epithelium caused by an impurity of malathion;Gandy;Amer. J. Pathol.,1983

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