Clinical and In Vitro Evidence That Subclinical Hemolysis Contributes to LVAD Thrombosis-Reference-Cited by-同舟云学术

Clinical and In Vitro Evidence That Subclinical Hemolysis Contributes to LVAD Thrombosis

Published:2018-03 Issue:3 Volume:105 Page:807-814
ISSN:0003-4975
Container-title:The Annals of Thoracic Surgery
language:en
Short-container-title:The Annals of Thoracic Surgery

Author:

Bartoli Carlo R.,Zhang David,Kang Jooeun,Hennessy-Strahs Samson,Restle David,Howard Jessica,Redline Gretchen,Bermudez Christian,Atluri Pavan,Acker Michael A.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Pulmonary and Respiratory Medicine,Surgery

Reference35 articles.

1. Diagnosis, nonsurgical management, and prevention of LVAD thrombosis;Bartoli;J Card Surg,2014

2. Diagnosis of hemolysis and device thrombosis with lactate dehydrogenase during left ventricular assist device support;Shah;J Heart Lung Transplant,2014

3. Hemolysate-mediated platelet aggregation: an additional risk mechanism contributing to thrombosis of continuous flow ventricular assist devices;Tran;Perfusion,2016

4. Acquired von Willebrand syndrome associated with left ventricular assist device;Nascimbene;Blood,2016

5. Pathologic von Willebrand factor degradation with a left ventricular assist device occurs via two distinct mechanisms: mechanical demolition and enzymatic cleavage;Bartoli;J Thorac Cardiovasc Surg,2015

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