Systemic treatment with neuropeptide Y receptor Y1-antagonist enhances atherosclerosis and stimulates IL-12 expression in ApoE deficient mice-Reference-Cited by-同舟云学术

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Systemic treatment with neuropeptide Y receptor Y1-antagonist enhances atherosclerosis and stimulates IL-12 expression in ApoE deficient mice

Published:2013-04 Issue:2 Volume:47 Page:67-73
ISSN:0143-4179
Container-title:Neuropeptides
language:en
Short-container-title:Neuropeptides

Author:

Jääskeläinen A.E.,Seppälä S.,Kakko T.,Jaakkola U.,Kallio J.

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Endocrine and Autonomic Systems,Neurology,Endocrinology,General Medicine

Reference50 articles.

1. Kinetic study of neuropeptide Y (NPY) proteolysis in blood and identification of NPY3-35: a new peptide generated by plasma kallikrein;Abid;J. Biol. Chem.,2009

2. Monocyte chemoattractant protein-1 accelerates atherosclerosis in apolipoprotein E-deficient mice;Aiello;Arterioscler. Thromb. Vasc. Biol.,1999

3. Altered endocannabinoid signalling after a high-fat diet in Apoe(−/−) mice: relevance to adipose tissue inflammation, hepatic steatosis and insulin resistance;Bartelt;Diabetologia,2011

4. Relevance of neuropeptide Y for the neuroimmune crosstalk;Bedoui;J. Neuroimmunol.,2003

5. The Y1 antagonist BIBP 3226 inhibits potentiation of methoxamine-induced vasoconstriction by neuropeptide Y;Bischoff;Naunyn Schmiedebergs Arch. Pharmacol.,1997

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