1. Angelotti, T.P. and Macdonald, R.L., Assembly of GABAA receptor subunits: α1β1 and α1β1Γ2s subunits produce unique ion channels with dissimilar single-channel properties, J. Neurosci., 13 (1993) 1429–1440.
2. Avanzini, G., De Curtis, M., Marescaux, C., Panzica, F., Spreafico, R. and Vergnes, M., Role of the thalamic reticular nucleus in the generation of rhythmic thalamo-cortical activities subserving spike and waves, J. Neural. Transm. Suppl., 35 (1992) 85–95.
3. Banerjee, P.K., Tillakaratne, N.J.K., Brailowsky, S., Olsen, R.W., Snead, O.C. and Tobin, A.J., Gamma-hydroxybutyrate (GHB)-induced absence-like seizures decrease the expression of GABAA receptor α4 mRNA in thalamus, Soc. Neurosci. Abstr., 21 (1995) 1592.
4. Berkovic, S.F., Childhood absence epilepsy and juvenile absence epilepsy. In: E. Wyllie (Ed.), The Treatment of Epilepsy: Principles and Practice, Lea and Febiger, Philadelphia, 1993b, pp. 547–551.
5. Berkovic, S.F., Generalized absence seizures. In: E. Wyllie (Ed.), The Treatment of Epilepsy: Principles and Practice, Lea and Febiger, Philadelphia, 1993a, pp. 401–410.