Transient increases in anti-aquaporin-4 antibody titers following rituximab treatment in neuromyelitis optica, in association with elevated serum BAFF levels-Reference-Cited by-同舟云学术

Transient increases in anti-aquaporin-4 antibody titers following rituximab treatment in neuromyelitis optica, in association with elevated serum BAFF levels

Published:2011-07 Issue:7 Volume:18 Page:997-998
ISSN:0967-5868
Container-title:Journal of Clinical Neuroscience
language:en
Short-container-title:Journal of Clinical Neuroscience

Author:

Nakashima Ichiro,Takahashi Toshiyuki,Cree Bruce A.C.,Kim Ho Jin,Suzuki Chihiro,Genain Claude P.,Vincent Thierry,Fujihara Kazuo,Itoyama Yasuto,Bar-Or Amit

Publisher

Elsevier BV

Subject

Physiology (medical),Clinical Neurology,Neurology,General Medicine,Surgery

Reference7 articles.

1. IgG marker of optic-spinal multiple sclerosis binds to the aquaporin-4 water channel;Lennon;J Exp Med,2005

2. Antibody to aquaporin-4 in the long-term course of neuromyelitis optica;Jarius;Brain,2008

3. Intrathecal pathogenic anti-aquaporin-4 antibodies in early neuromyelitis optica;Bennett;Annals of Neurology,2009

4. Treatment of neuromyelitis optica with rituximab: retrospective analysis of 25 patients;Jacob;Arch Neurol,2008

5. Rituximab mediates a strong elevation of B-cell-activating factor associated with increased pathogen-specific IgG but not autoantibodies in pemphigus vulgaris;Nagel;J Invest Dermatol,2009

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