The methylenetetrahydrofolate reductase 677TT genotype and folate intake interact to lower global leukocyte DNA methylation in young Mexican American women
Author:
Publisher
Elsevier BV
Subject
Nutrition and Dietetics,Endocrinology,Endocrinology, Diabetes and Metabolism
Reference17 articles.
1. DNA methylation, genomic silencing, and links to nutrition and cancer;McCabe;Nutr Rev,2005
2. A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase;Frosst;Nat Genet,1995
3. Methylenetetrahydrofolate reductase 677C→T variant modulates folate status response to controlled folate intakes in young women;Guinotte;J Nutr,2003
4. Folate and genetics;Rozen;J Food Sc,2004
5. Moderate folate depletion increases plasma homocysteine and decreases lymphocyte DNA methylation in postmenopausal women;Jacob;J Nutr,1998
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1. The 677C > T variant in methylenetetrahydrofolate reductase causes morphological and functional cerebrovascular deficits in mice;Journal of Cerebral Blood Flow & Metabolism;2022-09-01
2. The 677C>T variant in methylenetetrahydrofolate reductase causes morphological and functional cerebrovascular deficits in mice;2021-12-16
3. Prenatal Multivitamin Use and MTHFR Genotype Are Associated with Newborn Cord Blood DNA Methylation;International Journal of Environmental Research and Public Health;2020-12-09
4. Global DNA Methylation as a Potential Underlying Mechanism of Congenital Disease Development;DNA Methylation Mechanism;2020-07-01
5. Association of genetic and epigenetic variants in one-carbon metabolism gene with folate treatment response in hyperhomocysteinaemia;European Journal of Clinical Nutrition;2020-03-20
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