Sodium Channels
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Publisher
Elsevier
Reference220 articles.
1. Nav1.7 is the predominant sodium channel in rodent olfactory sensory neurons;Ahn;Mol. Pain,2011
2. The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways;Akopian;Nat. Neurosci.,1999
3. FHF1 (FGF12) epileptic encephalopathy;Al-Mehmadi;Neurol. Genet.,2016
4. Inactivation of open and closed sodium channels determined separately;Aldrich;Cold Spring Harbor Symp. Quant. Biol.,1983
5. Subtype-selective small molecule inhibitors reveal a fundamental role for Nav1.7 in nociceptor electrogenesis, axonal conduction and presynaptic release;Alexandrou;PLoS One,2016
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1. Analgesic effect of Botulinum toxin in neuropathic pain is sodium channel independent;Neuropharmacology;2024-08
2. The pain target NaV1.7 is expressed late during human iPS cell differentiation into sensory neurons as determined in high-resolution imaging;Pflügers Archiv - European Journal of Physiology;2024-03-27
3. A modelling study to dissect the potential role of voltage-gated ion channels in activity-dependent conduction velocity changes as identified in small fiber neuropathy patients;Frontiers in Computational Neuroscience;2023-12-14
4. Biophysics of sodium channels during subthreshold depolarizationin vitroandin silico;2023-09-06
5. Cold and warmth intensify pain-linked sodium channel gating effects and persistent currents;Journal of General Physiology;2023-08-02
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