Constitutive Activity of the Acetylcholine-Activated Potassium Current IK,ACh in Cardiomyocytes-Reference-Cited by-同舟云学术

Constitutive Activity of the Acetylcholine-Activated Potassium Current IK,ACh in Cardiomyocytes

Published:2014 Issue: Volume: Page:393-409
ISSN:1054-3589
Container-title:Pharmacology & Therapeutics of Constitutively Active Receptors
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Author:

Voigt Niels,Abu-Taha Issam,Heijman Jordi,Dobrev Dobromir

Publisher

Elsevier

Reference71 articles.

1. Nucleoside diphosphate kinase B is a novel receptor-independent activator of G-protein signaling in clinical and experimental atrial fibrillation;Abu-Taha;Naunyn-Schmiedeberg's Archives of Pharmacology,2013

2. G protein-activated (GIRK) current in rat ventricular myocytes is masked by constitutive inward rectifier current (IK1);Beckmann;Cellular Physiology and Biochemistry,2008

3. Tissue-specific effects of acetylcholine in the canine heart;Calloe;American Journal of Physiology. Heart and Circulatory Physiology,2013

4. Kir3-based inward rectifier potassium current: Potential role in atrial tachycardia remodeling effects on atrial repolarization and arrhythmias;Cha;Circulation,2006

5. Phosphatidylinositol 4,5-bisphosphate is acting as a signal molecule in α1-adrenergic pathway via the modulation of acetylcholine-activated K+ channels in mouse atrial myocytes;Cho;The Journal of Biological Chemistry,2001

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