The History, Biology, and Pathophysiology of Apparent Mineralocorticoid Excess
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Elsevier
Reference11 articles.
1. A genetic defect in cortisol metabolism as the basis for the syndrome of apparent mineralocorticoid excess;New,1982
2. Renal mineralocorticoid receptors and hippocampal corticosterone binding species have identical intrinsic steroid specificity;Krozowski;Proc Natl Acad Sci USA,1983
3. Differential binding of NAD+ and NADH allows the transcriptional corepressor carboxyl-terminal binding protein to serve as a metabolic sensor;Fjeld;Proc Natl Acad Sci USA,2003
4. Glucocorticoids activate cardiac mineralocorticoid receptors during experimental myocardial infarction;Mihailidou;Hypertension,2009
5. Mineralocorticoid receptors: evolutionary and pathophysiological considerations;Kassahn;Endocrinology,2011
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1. Apparent Mineralocorticoid Excess in the Pediatric Population: Report of a Novel Pathogenic Variant of the 11 beta-HSD2 Gene and Systematic Review of the Literature;PEDIATR ENDOCR REV P;2019
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