Rickets Due to Hereditary Abnormalities of Vitamin D Synthesis or Action-Reference-Cited by-同舟云学术

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Rickets Due to Hereditary Abnormalities of Vitamin D Synthesis or Action

Published:2012 Issue: Volume: Page:679-698
ISSN:
Container-title:Pediatric Bone
language:
Short-container-title:

Author:

Portale Anthony A.,Perwad Farzana,Miller Walter L.

Publisher

Elsevier

Reference159 articles.

1. The 25-hydroxyvitamin D 1-a-hydroxylase;Henry,2005

2. 1,25-dihydroxyvitamin D3 production by human keratinocytes;Bikle;J Clin Invest,1986

3. Metabolism of 25-hydroxyvitamin D3 by cultured pulmonary alveolar macrophages in sarcoidosis;Adams;J Clin Invest,1983

4. Human bone cells in culture metabolize 25-hydroxyvitamin D3 to 1,25-dihydroxyvitamin D3 and 24,25-dihydroxyvitamin D3;Howard;J Biol Chem,1981

5. Characterization and regulation of the vitamin D hydroxylases;Armbrecht;J Steroid Biochem Mol Biol,1992

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Cinacalcet treatment experience in hereditary vitamin D resistant rickets;Journal of Pediatric Endocrinology and Metabolism;2020-02-25

2. Vitamin D-Dependent Rickets Type 1B (25-Hydroxylase Deficiency): A Rare Condition or a Misdiagnosed Condition?;Journal of Bone and Mineral Research;2017-07-13

3. FGF-23 Regulates CYP27B1 Transcription in the Kidney and in Extra-Renal Tissues;PLoS ONE;2013-09-03

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