Alpha-synuclein immunoreactivity of huntingtin polyglutamine aggregates in striatum and cortex of Huntington's disease patients and transgenic mouse models-Reference-Cited by-同舟云学术

Alpha-synuclein immunoreactivity of huntingtin polyglutamine aggregates in striatum and cortex of Huntington's disease patients and transgenic mouse models

Published:2000-07 Issue:1 Volume:289 Page:29-32
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Charles Vinod,Mezey Eva,Reddy P.Hemachandra,Dehejia Anindya,Young Theresa A,Polymeropoulos Mihail H,Brownstein Michael J,Tagle Danilo A

Publisher

Elsevier BV

Subject

General Neuroscience

Reference17 articles.

1. Fast transport and retrograde movement of huntingtin and HAP 1 in axons;Block-Galarza;NeuroReport,1997

2. Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human huntington disease gene;Cha;Proc. Natl. Acad. Sci. USA,1998

3. The synucleins: a family of proteins involved in synaptic function, plasticity, neurodegeneration and disease;Clayton;Trends Neurosci.,1998

4. α-Synuclein overexpression promotes aggregation of mutant huntingtin;Furlong;Biochem. J.,2000

5. Ultrastructural localization and progressive formation of neuropil aggregates in Huntington's disease transgenic mice;Li;Hum. Mol. Genet.,1999

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