Activation of c-jun N-terminal kinase/stress-activated protein kinase and the decreased ratio of Bcl-2 to Bax are associated with the auto-oxidized dopamine-induced apoptosis in PC12 cells-Reference-Cited by-同舟云学术

Activation of c-jun N-terminal kinase/stress-activated protein kinase and the decreased ratio of Bcl-2 to Bax are associated with the auto-oxidized dopamine-induced apoptosis in PC12 cells

Published:1998-10 Issue:1 Volume:256 Page:37-40
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Kang Chi-Dug,Jang Jung-Hee,Kim Kwang-Woon,Lee Heon-Jin,Jeong Choon-Sik,Kim Cheol-Min,Kim Sun-Hee,Chung Byung-Seon

Publisher

Elsevier BV

Subject

General Neuroscience

Reference20 articles.

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3. Altered expression of Bcl-2, Bcl-X, Bax, and c-Fos colocalizes with DNA fragmentation and ischemic cell damage following middle cerebral artery occlusion in rats;Gillardon;Brain. Res. Mol. Brain. Res.,1996

4. Auto-oxidation versus covalent binding of quinones as the mechanism of toxicity of dopamine, 6-hydroxydopamine, and related compounds toward C1300 neuroblastoma cells in vitro;Graham;Mol. Pharmacol.,1978

5. Enzymatic oxidation of dopamine: the role of prostaglandin H synthase;Hastings;J. Neurochem.,1995

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