Δ9-Tetrahydrocannabinol excites rat VTA dopamine neurons through activation of cannabinoid CB1 but not opioid receptors

Author:

French Edward D

Publisher

Elsevier BV

Subject

General Neuroscience

Reference20 articles.

1. A comparison of some pharmacological actions of morphine and Δ9-tetrahydrocannabinol in the mouse;Bloom;Psychopharmacology,1978

2. Bloom, A.S. and Hillard, C.J., Cannabinoids, neurotransmitters, receptors, and brain membranes. In D.J. Harvey (Ed.), Marijuana '84, IRL Press, Oxford, 1985, pp. 217–231.

3. Dopaminergic neurons: effects of antipsychotic drugs and amphetamine on single cell activity;Bunney;J. Pharmacol. Exp. Ther.,1973

4. Δ9-Tetrahydrocannabinol produces naloxone-blockable enhancement of presynaptic basal dopamine efflux in nucleus accumbens of conscious, freely-moving rats as measured by intracerebral microdialysis;Chen;Psychopharmacology,1990

5. Competitive NMDA receptor antagonists attenuate phencyclidine-induced excitations of A10 dopamine neurons;French;Eur. J. Pharmacol.,1992

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