Functions of p53 suppress critical consequences of damage and repair in the initiation of cancer
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Cell Biology,Oncology
Reference13 articles.
1. Selective multiplication of dihydrofolate reductase genes in methotrexate-resistant variants of cultured murine cells.
2. DNA double-strand break repair proteins are required to cap the ends of mammalian chromosomes
3. Metaphase Chromosome Anomaly: Association with Drug Resistance and Cell-Specific Products
4. Novel DNA rearrangements are associated with dihydrofolate reductase gene amplification.
5. p63 and p73 are required for p53-dependent apoptosis in response to DNA damage
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