Tumor-Suppressor Inactivation of GDF11 Occurs by Precursor Sequestration in Triple-Negative Breast Cancer-Reference-Cited by-同舟云学术

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Tumor-Suppressor Inactivation of GDF11 Occurs by Precursor Sequestration in Triple-Negative Breast Cancer

Published:2017-11 Issue:4 Volume:43 Page:418-435.e13
ISSN:1534-5807
Container-title:Developmental Cell
language:en
Short-container-title:Developmental Cell

Author:

Bajikar Sameer S.,Wang Chun-Chao,Borten Michael A.,Pereira Elizabeth J.,Atkins Kristen A.,Janes Kevin A.

Funder

American Cancer Society

NIH

UVA Cancer Center

Ivy Foundation

David and Lucile Packard Foundation

National Science Foundation

University of Virginia

Biorepository and Tissue Research Facility

Molecular Imaging Core

University of Virginia Cancer Center

Publisher

Elsevier BV

Subject

Developmental Biology,Cell Biology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology

Reference140 articles.

1. Different array CGH profiles within hereditary breast cancer tumors associated to BRCA1 expression and overall survival;Alvarez;BMC Cancer,2016

2. Growth differentiation factor 11 signals through the transforming growth factor-beta receptor ALK5 to regionalize the anterior-posterior axis;Andersson;EMBO Rep.,2006

3. Membrane-anchored and soluble forms of betaglycan, a polymorphic proteoglycan that binds transforming growth factor-beta;Andres;J. Cell Biol.,1989

4. Role of PCSK5 expression in mouse ovarian follicle development: identification of the inhibin alpha- and beta-subunits as candidate substrates;Antenos;PLoS One,2011

5. Parameterizing cell-to-cell regulatory heterogeneities via stochastic transcriptional profiles;Bajikar;Proc. Natl. Acad. Sci. USA,2014

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