Notch Ligand Endocytosis Generates Mechanical Pulling Force Dependent on Dynamin, Epsins, and Actin-Reference-Cited by-同舟云学术

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Notch Ligand Endocytosis Generates Mechanical Pulling Force Dependent on Dynamin, Epsins, and Actin

Published:2012-06 Issue:6 Volume:22 Page:1299-1312
ISSN:1534-5807
Container-title:Developmental Cell
language:en
Short-container-title:Developmental Cell

Author:

Meloty-Kapella Laurence,Shergill Bhupinder,Kuon Jane,Botvinick Elliot,Weinmaster Gerry

Publisher

Elsevier BV

Subject

Developmental Biology,Cell Biology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology

Reference59 articles.

1. Differential requirements for actin during yeast and mammalian endocytosis;Aghamohammadzadeh;Nat. Cell Biol.,2009

2. Epsin N-terminal homology domains perform an essential function regulating Cdc42 through binding Cdc42 GTPase-activating proteins;Aguilar;Proc. Natl. Acad. Sci. USA,2006

3. The functions of auxilin and Rab11 in Drosophila suggest that the fundamental role of ligand endocytosis in notch signaling cells is not recycling;Banks;PLoS ONE,2011

4. Selective use of ADAM10 and ADAM17 in activation of Notch1 signaling;Bozkulak;Mol. Cell. Biol.,2009

5. Regulation of Hip1r by epsin controls the temporal and spatial coupling of actin filaments to clathrin-coated pits;Brady;J. Cell Sci.,2010

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