Combined Deficiency of Tet1 and Tet2 Causes Epigenetic Abnormalities but Is Compatible with Postnatal Development-Reference-Cited by-同舟云学术

Combined Deficiency of Tet1 and Tet2 Causes Epigenetic Abnormalities but Is Compatible with Postnatal Development

Published:2013-02 Issue:3 Volume:24 Page:310-323
ISSN:1534-5807
Container-title:Developmental Cell
language:en
Short-container-title:Developmental Cell

Author:

Dawlaty Meelad M.,Breiling Achim,Le Thuc,Raddatz Günter,Barrasa M. Inmaculada,Cheng Albert W.,Gao Qing,Powell Benjamin E.,Li Zhe,Xu Mingjiang,Faull Kym F.,Lyko Frank,Jaenisch Rudolf

Publisher

Elsevier BV

Subject

Developmental Biology,Cell Biology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology

Reference36 articles.

1. A bivalent chromatin structure marks key developmental genes in embryonic stem cells;Bernstein;Cell,2006

2. Hydroxylation of 5-methylcytosine by TET2 maintains the active state of the mammalian HOXA cluster;Bocker;Nat. Commun.,2012

3. Uncovering the role of 5-hydroxymethylcytosine in the epigenome;Branco;Nat. Rev. Genet.,2011

4. TET2 promotes histone O-GlcNAcylation during gene transcription;Chen;Nature,2012

5. Tet1 is dispensable for maintaining pluripotency and its loss is compatible with embryonic and postnatal development;Dawlaty;Cell Stem Cell,2011

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2. TET enzyme driven epigenetic reprogramming in early embryos and its implication on long-term health;Frontiers in Cell and Developmental Biology;2024-08-01

3. TET1 displays catalytic and non-catalytic functions in the adult mouse cortex;Epigenetics;2024-07-06

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