A Yap-Myc-Sox2-p53 Regulatory Network Dictates Metabolic Homeostasis and Differentiation in Kras-Driven Pancreatic Ductal Adenocarcinomas-Reference-Cited by-同舟云学术

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A Yap-Myc-Sox2-p53 Regulatory Network Dictates Metabolic Homeostasis and Differentiation in Kras-Driven Pancreatic Ductal Adenocarcinomas

Published:2019-10 Issue:1 Volume:51 Page:113-128.e9
ISSN:1534-5807
Container-title:Developmental Cell
language:en
Short-container-title:Developmental Cell

Author:

Murakami Shigekazu,Nemazanyy Ivan,White Shannon M.,Chen Hengye,Nguyen Chan D.K.,Graham Garrett T.,Saur Dieter,Pende Mario,Yi Chunling

Funder

NIH

Sher grant

Cancer Center Support grant

Publisher

Elsevier BV

Subject

Developmental Biology,Cell Biology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology

Reference72 articles.

1. Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma;Aguirre;Genes Dev.,2003

2. Ptf1a-mediated control of Dll1 reveals an alternative to the lateral inhibition mechanism;Ahnfelt-Rønne;Development,2012

3. Oct4 switches partnering from Sox2 to Sox17 to reinterpret the enhancer code and specify endoderm;Aksoy;EMBO J.,2013

4. Genomic analyses identify molecular subtypes of pancreatic cancer;Bailey;Nature,2016

5. SOX2 is an amplified lineage-survival oncogene in lung and esophageal squamous cell carcinomas;Bass;Nat. Genet.,2009

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