A Senescence Program Controlled by p53 and p16INK4a Contributes to the Outcome of Cancer Therapy-Reference-Cited by-同舟云学术

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A Senescence Program Controlled by p53 and p16INK4a Contributes to the Outcome of Cancer Therapy

Published:2002-05 Issue:3 Volume:109 Page:335-346
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Schmitt Clemens A.,Fridman Jordan S.,Yang Meng,Lee Soyoung,Baranov Eugene,Hoffman Robert M.,Lowe Scott W.

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference41 articles.

1. The c-myc oncogene driven by immunoglobulin enhancers induces lymphoid malignancy in transgenic mice;Adams;Nature,1985

2. Involvement of the cyclin-dependent kinase inhibitor p16INK4a in replicative senescence of normal human fibroblasts;Alcorta;Proc. Natl. Acad. Sci. USA,1996

3. Dual inactivation of RB and p53 pathways in RAS-induced melanomas;Bardeesy;Mol. Cell. Biol.,2001

4. Genetic determinants of response to chemotherapy in transgenic mouse mammary and salivary tumors;Bearss;Oncogene,2000

5. Apoptosis, p53, and tumor cell sensitivity to anticancer agents;Brown;Cancer Res.,1999

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