Anosmin-1, Defective in the X-Linked Form of Kallmann Syndrome, Promotes Axonal Branch Formation from Olfactory Bulb Output Neurons

Author:

Soussi-Yanicostas Nadia,de Castro Fernando,Julliard A.Karyn,Perfettini Isabelle,Chédotal Alain,Petit Christine

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference54 articles.

1. Characterization of the two zebrafish orthologues of the KAL-1 gene underlying X chromosome-linked Kallmann syndrome;Ardouin;Mech. Dev.,2000

2. Slit proteins bind Robo receptors and have an evolutionarily conserved role in repulsive axon guidance;Brose;Cell,1999

3. Heparan sulfate proteoglycan-dependent induction of axon branching and axon misrouting by the Kallmann syndrome gene kal-1;Buelow;Proc. Natl. Acad. Sci. USA,2002

4. Semaphorins III and IV repel hippocampal axons via two distinct receptors;Chédotal;Development,1998

5. Effects of brain-derived neurotrophic factor on optic axon branching and remodelling in vivo;Cohen-Cory;Nature,1995

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