Xingnaojing ameliorates synaptic plasticity and memory deficits in an Aβ1–42 induced mouse model of Alzheimer's disease-Reference-Cited by-同舟云学术

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Xingnaojing ameliorates synaptic plasticity and memory deficits in an Aβ1–42 induced mouse model of Alzheimer's disease

Published:2020-08 Issue:4 Volume:143 Page:245-254
ISSN:1347-8613
Container-title:Journal of Pharmacological Sciences
language:en
Short-container-title:Journal of Pharmacological Sciences

Author:

Liu Yi,Xu Siyi,Bian Huijie,Qian Yi,Li Huiya,Shu Shu,Chen Jiang,Cao Xiang,Gu Yue,Jin Jiali,Zhang Xi,Xu Yun,Zhu Xiaolei

Funder

National Natural Science Foundation of China

National Key Research and Development Program of China

Key Research and Development Program of Jiangsu Province of China

Jiangsu Province Key Medical Discipline

Publisher

Elsevier BV

Subject

Pharmacology,Molecular Medicine

Reference35 articles.

1. Alzheimer's disease: a Journey from amyloid peptides and oxidative stress, to biomarker technologies and disease prevention strategies-gains from AIBL and DIAN cohort studies;Martins;J Alzheimers Dis,2018

2. The toxic Abeta oligomer and Alzheimer's disease: an emperor in need of clothes;Benilova;Nat Neurosci,2012

3. The amyloid hypothesis of Alzheimer's disease at 25 years;Selkoe;EMBO Mol Med,2016

4. Fyn-tau-amyloid: a toxic triad;Haass;Cell,2010

5. Spines, plasticity, and cognition in Alzheimer's model mice;Spires-Jones;Neural Plast,2012

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