A negative elongation factor E inhibits white spot syndrome virus replication by suppressing promoter activity of the viral immediate early genes in red claw crayfish Cherax quadricarinatus

Author:

Gao Yan,Liu Ling-ke,Wang Ke-jian,Liu Hai-peng

Funder

Natural Science Foundation of China

Fundamental Research Funds for the Central Universities

Publisher

Elsevier BV

Subject

Developmental Biology,Immunology

Reference40 articles.

1. NELF-E is recruited to DNA double-strand break sites to promote transcriptional repression and repair;Awwad;EMBO Rep.,2017

2. Role of Penaeus monodon Kruppel-like factor (PmKLF) in infection by white spot syndrome virus;Chang;Dev. Comp. Immunol.,2012

3. White spot syndrome virus entry is dependent on multiple endocytic routes and strongly facilitated by Cq-GABARAP in a CME-dependent manner;Chen;Sci. Rep.,2016

4. A review on the morphology, molecular characterization, morphogenesis and pathogenesis of white spot syndrome virus;Escobedo-Bonilla;J. Fish. Dis.,2008

5. Defining mechanisms that regulate RNA polymerase II transcription in vivo;Fuda;Nature,2009

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