Expansion of a suppressor T-cell population associated with the hyper-IgM syndrome and generalized lymphadenopathy-Reference-Cited by-同舟云学术

Expansion of a suppressor T-cell population associated with the hyper-IgM syndrome and generalized lymphadenopathy

Published:1991-07 Issue:1 Volume:60 Page:118-127
ISSN:0090-1229
Container-title:Clinical Immunology and Immunopathology
language:en
Short-container-title:Clinical Immunology and Immunopathology

Author:

Hoeger Peter H.,Mayer Lloyd

Publisher

Elsevier BV

Subject

Immunology,Pathology and Forensic Medicine,Immunology and Allergy

Reference20 articles.

1. Recurrent bacterial infections and dysgammaglobulinemia: Deficiency of 7S gammaglobulins in the presence of elevated 19S gammaglobulins;Rosen;Pediatrics,1961

2. Hyper-immunoglobulin M immunodeficiency (dysgammaglobulinemia): Presence of immunoglobulin M-secreting plasmacytoid cells in peripheral blood and failure of immunoglobulin M- immunoglobulin G-switch in B-cell differentiation;Geha;J. Clin. Invest.,1979

3. Abnormal B-cell differentiation and variable increased T-cell suppression in immunodeficiency with hyper-IgM;Krantman;Clin. Exp. Immunol.,1980

4. Immunologic studies of three family members with the immunodeficiency with hyper-IgM syndrome;Brahmi;J. Clin. Immunol.,1983

5. Hyper-IgM immunodeficiency: A primary dysfunction of B-lymphocyte isotype switching;Levitt;J. Clin. Invest.,1983

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2. Immunoglobulin class switch recombination deficiency type 1 or CD40 ligand deficiency: from bedside to bench and back again;Expert Review of Clinical Immunology;2013-11-26

3. Genetically acquired class-switch recombination defects: the multi-faced hyper-IgM syndrome;Immunology Letters;2005-02

4. Differential activation requirements of isotype-switched B cells;European Journal of Immunology;1996-08