Stress- and diet-induced fat gain is controlled by NPY in catecholaminergic neurons-Reference-Cited by-同舟云学术

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Stress- and diet-induced fat gain is controlled by NPY in catecholaminergic neurons

Published:2014-08 Issue:5 Volume:3 Page:581-591
ISSN:2212-8778
Container-title:Molecular Metabolism
language:en
Short-container-title:Molecular Metabolism

Author:

Zhang Lei,Lee I-Chieh J.,Enriquez Rondaldo F.,Lau Jackie,Vähätalo Laura H.,Baldock Paul A.,Savontaus Eriika,Herzog Herbert

Funder

National Health and Medical Research Council

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference47 articles.

1. The neuropeptide Y system: pathophysiological and therapeutic implications in obesity and cancer;Zhang;Pharmacology & Therapeutics,2011

2. NPY and cohorts in regulating appetite, obesity and metabolic syndrome: beneficial effects of gene therapy;Kalra;Neuropeptides,2004

3. An arcuato-paraventricular and -dorsomedial hypothalamic neuropeptide Y-containing system which lacks noradrenaline in the rat;Bai;Brain Research,1985

4. Arcuate NPY controls sympathetic output and BAT function via a relay of tyrosine hydroxylase neurons in the PVN;Shi;Cell Metabolism,2013

5. Colocalization of neuropeptide Y immunoreactivity in brainstem catecholaminergic neurons that project to the paraventricular nucleus of the hypothalamus;Sawchenko;Journal of Comparative Neurology,1985

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