Deletion of Six3 in post-proliferative neurons produces weakened SCN circadian output, improved metabolic function, and dwarfism in male mice
Author:
Funder
NIH
Office of Naval Research
Department of Veterans Affairs
NICHD
USDA Agricultural Research Service
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
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Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. The transcription factor VAX1 in VIP neurons of the suprachiasmatic nucleus impacts circadian rhythm generation, depressive-like behavior, and the reproductive axis in a sex-specific manner in mice;Frontiers in Endocrinology;2023-12-22
2. Heterozygous variants in SIX3 and POU1F1 cause pituitary hormone deficiency in mouse and man;Human Molecular Genetics;2022-08-11
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