N-terminal transactivation function, AF-1, of estrogen receptor alpha controls obesity through enhancement of energy expenditure
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference32 articles.
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3. Prevention of obesity and insulin resistance by estrogens requires ERα activation function-2 (ERαAF-2), whereas ERαAF-1 is dispensable;Handgraaf;Diabetes,2013
4. The transactivating function 1 of estrogen receptor α is dispensable for the vasculoprotective actions of 17β-estradiol;Billon-Galés;Proceedings of the National Academy of Sciences,2009
5. Activation function 2 (AF2) of estrogen receptor-alpha is required for the atheroprotective action of estradiol but not to accelerate endothelial healing;Billon-Galés;Proceedings of the National Academy of Sciences of the United States of America,2011
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