Mice lacking neutral amino acid transporter B0AT1 (Slc6a19) have elevated levels of FGF21 and GLP-1 and improved glycaemic control-Reference-Cited by-同舟云学术

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Mice lacking neutral amino acid transporter B0AT1 (Slc6a19) have elevated levels of FGF21 and GLP-1 and improved glycaemic control

Published:2015-05 Issue:5 Volume:4 Page:406-417
ISSN:2212-8778
Container-title:Molecular Metabolism
language:en
Short-container-title:Molecular Metabolism

Author:

Jiang Yang,Rose Adam J.,Sijmonsma Tjeerd P.,Bröer Angelika,Pfenninger Anja,Herzig Stephan,Schmoll Dieter,Bröer Stefan

Funder

Sanofi-Aventis, Germany

EFSD/Lilly European Diabetes Research Programme

Helmholtz Programs

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference64 articles.

1. Role of dietary proteins and amino acids in the pathogenesis of insulin resistance;Tremblay;Annual Review of Nutrition,2007

2. Mechanism of amino acid-induced skeletal muscle insulin resistance in humans;Krebs;Diabetes,2002

3. Hyperaminoacidaemia reduces insulin-mediated glucose disposal in healthy man;Tessari;Diabetologia,1985

4. Inappropriate activation of the TSC/Rheb/mTOR/S6K cassette induces IRS1/2 depletion, insulin resistance, and cell survival deficiencies;Shah;Current Biology,2004

5. Regulation of insulin sensitivity by serine/threonine phosphorylation of insulin receptor substrate proteins IRS1 and IRS2;Copps;Diabetologia,2012

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