RIPK3 promotes islet amyloid-induced β-cell loss and glucose intolerance in a humanized mouse model of type 2 diabetes-Reference-Cited by-同舟云学术

RIPK3 promotes islet amyloid-induced β-cell loss and glucose intolerance in a humanized mouse model of type 2 diabetes

Published:2024-02 Issue: Volume:80 Page:101877
ISSN:2212-8778
Container-title:Molecular Metabolism
language:en
Short-container-title:Molecular Metabolism

Author:

Mukherjee Noyonika,Contreras Christopher J.,Lin Li,Colglazier Kaitlyn A.,Mather Egan G.,Kalwat Michael A.,Esser Nathalie,Kahn Steven E.,Templin Andrew T.^ORCID

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

US Department of Veterans Affairs

Ralph W and Grace M Showalter Research Trust Fund

Publisher

Elsevier BV

Reference61 articles.

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3. Islet amyloid: a long-recognized but underappreciated pathological feature of type 2 diabetes;Kahn;Diabetes,1999

4. β-cell loss and β-cell apoptosis in human type 2 diabetes are related to islet amyloid deposition;Jurgens;Am J Pathol,2011

5. Islet amyloid formation is an important determinant for inducing islet inflammation in high-fat-fed human IAPP transgenic mice;Meier;Diabetologia,2014

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