Deletion of Ascl1 in pancreatic β-cells improves insulin secretion, promotes parasympathetic innervation, and attenuates dedifferentiation during metabolic stress-Reference-Cited by-同舟云学术

Deletion of Ascl1 in pancreatic β-cells improves insulin secretion, promotes parasympathetic innervation, and attenuates dedifferentiation during metabolic stress

Published:2023-12 Issue: Volume:78 Page:101811
ISSN:2212-8778
Container-title:Molecular Metabolism
language:en
Short-container-title:Molecular Metabolism

Author:

Osipovich Anna B.,Zhou Frank Y.,Chong Judy J.,Trinh Linh T.,Cottam Mathew A.,Shrestha Shristi,Cartailler Jean-Philippe,Magnuson Mark A.^ORCID

Funder

National Institutes of Health

National Eye Institute

National Cancer Institute

NIDDK

Vanderbilt University

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference78 articles.

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4. Alterations in beta-cell calcium dynamics and efficacy outweigh islet mass adaptation in compensation of insulin resistance and prediabetes onset;Chen;Diabetes,2016

5. Pancreatic beta cell dedifferentiation as a mechanism of diabetic beta cell failure;Talchai;Cell,2012

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1. Ca2+ signaling and metabolic stress-induced pancreatic β-cell failure;Frontiers in Endocrinology;2024-07-02

2. PAX4 gene delivery improves β-cell function in human islets of Type II diabetes;Regenerative Medicine;2024-05-03