Formation of prostaglandin D2 during endoperoxide-induced platelet aggregation

Author:

Bryan Smith J.,Ingerman Carol M.,Silver Melvin J.

Publisher

Elsevier BV

Subject

Hematology

Reference11 articles.

1. Formation of an intermediate in prostaglandin biosynthesis and its association with the platelet release reaction;Smith;J. Clin. Invest.,1974

2. Isolation and structure of two prostaglandin endoperoxides that cause platelet aggregation;Hamberg,1974

3. An endoperoxide aggregator (LASS), formed in platelets in response to thrombotic stimuli — purification, identification and unique biological significance;Willis;Prostaglandins,1974

4. Physiological role of an endoperoxide in human platelets: Hemostatic defect due to platelet cyclo-oxygenase deficiency;Malmsten,1975

5. Prostaglandin D2 inhibits the aggregation of human platelets;Smith;Thrombosis Research,1974

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1. Cytotoxicity of activated platelets to autologous red blood cells;British Journal of Haematology;1992-09

2. The effect of CV-4151, a selective inhibitor of thromboxane synthetase, on prostanoid formation and platelet aggregation in humans;Cardiovascular Drugs and Therapy;1988-12

3. Separation of thromboxane B2 by high-performance liquid chromatography;Journal of Chromatography B: Biomedical Sciences and Applications;1987-01

4. Prostaglandins, Thromboxanes and Platelet Function;Prostacyclin and Its Stable Analogue Iloprost;1987

5. Ihibitory effect of prostaglandin D2 on dna synthesis in nuclei;Prostaglandins, Leukotrienes and Medicine;1986-08

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