Release of prostacyclin into the bloodstream and its exhaustion in humans after local blood flow changes (ischemia and venous stasis)
Author:
Publisher
Elsevier BV
Subject
Hematology
Reference21 articles.
1. An enzyme isolated from arteries transforms prostaglandin endoperioxides to an unstable substance that inhibits platelet aggregation;Moncada;Nature.,1976
2. Differential formation of prostacyclin (PGX or PGI2) by layers of the arterial wall;Moncada,1977
3. Human arterial and venous tissue generate prostacyclin (Prostaglandin X), a potent inhibitor of platelet aggregation;Moncada;Lancet.,1977
4. Prostacyclin (PGX) is the endogenous metabolite responsible for relaxation of coronary induced by arachidonic acid;Dusting;Prostaglandins.,1977
5. The chemical structure of prostaglandin X (Prostacyclin);Johnson;Prostaglandins.,1976
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1. Thromboxane A2 and Prostacyclin Release in Bleeding Time Blood during Primary Haemostasis in Healthy Individuals;Acta Medica Scandinavica;2009-04-24
2. Elevated urinary levels of thromboxane and prostacyclin metabolites in sickle cell disease reflects activated platelets in the circulation;British Journal of Haematology;1994-07
3. Effects of exercise training on the biosynthesis of prostacyclin and thromboxane in rats;Acta Physiologica Scandinavica;1993-01
4. Repeated sympathetic stimuli elicit the decline and disappearance of prostaglandin modulation and an increase of vascular resistance in humans.;Circulation Research;1990-09
5. The effect of exercise on bleeding time and local production of prostacyclin and thromboxane;European Journal of Applied Physiology and Occupational Physiology;1989-12
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