ADP-, PAF- and adrenaline-induced platelet aggregation and thromboxane formation are not affected by a thromboxane receptor antagonist at physiological external Ca++ concentrations-Reference-Cited by-同舟云学术

ADP-, PAF- and adrenaline-induced platelet aggregation and thromboxane formation are not affected by a thromboxane receptor antagonist at physiological external Ca++ concentrations

Published:1994-08 Issue:3 Volume:75 Page:233-242
ISSN:0049-3848
Container-title:Thrombosis Research
language:en
Short-container-title:Thrombosis Research

Author:

Bretschneider Ellen,Glusa Erika,Schrör Karsten

Publisher

Elsevier BV

Subject

Hematology

Reference29 articles.

1. Aggregation of platelets by adenosine diphosphate and its reversal;Born;Nature,1962

2. Role of cytoplasmic and releasable ADP in platelet aggregation induced by laminar shear stress;Moritz;J. Lab. Clin. Med.,1983

3. The stimulation of arachidonic acid metabolism in human platelets by hydrodynamic stress;Rajagopalan;Biochim. Biophys. Acta,1988

4. Synergistic responses in human platelets;Thompson,1986

5. Thromboxane A2 causes feedback amplification involving extensive thromboxane A2 formation upon close contact of human platelets in media with a low concentration of ionized calcium;Packham;Blood,1987

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