Neuroprotection by JM-1232(−) against oxygen–glucose deprivation-induced injury in rat hippocampal slice culture-Reference-Cited by-同舟云学术

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Neuroprotection by JM-1232(−) against oxygen–glucose deprivation-induced injury in rat hippocampal slice culture

Published:2015-01 Issue: Volume:1594 Page:52-60
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Ogura Takahiro,Hamada Tsuyoshi,Matsui Toshiyasu,Tanaka Shinji,Okabe Shigeo,Kazama Tomiei,Kobayashi Yasushi

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference54 articles.

1. Midazolam improves electrophysiologic recovery after anoxia and reduces the changes in ATP levels and calcium influx during anoxia in the rat hippocampal slice;Abramowicz;Anesthesiology,1991

2. Central nervous system complications of cardiac surgery;Arrowsmith;Br. J. Anaesth.,2000

3. Protective role of neuronal KATP channels in brain hypoxia;Ballanyi;J. Exp. Biol.,2004

4. High-dose propofol triggers short-term neuroprotection and long-term neurodegeneration in primary neuronal cultures from rat embryos;Berns;J. Int. Med. Res.,2009

5. γ-aminobutyric acid-A receptors contribute to isoflurane neuroprotection in organotypic hippocampal cultures;Bickler;Anesth. Analg.,2003

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