Characterization of DNA hypermethylation in the cerebellum of c9FTD/ALS patients-Reference-Cited by-同舟云学术

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Characterization of DNA hypermethylation in the cerebellum of c9FTD/ALS patients

Published:2014-10 Issue: Volume:1584 Page:15-21
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Belzil Veronique V.,Bauer Peter O.,Gendron Tania F.,Murray Melissa E.,Dickson Dennis,Petrucelli Leonard

Funder

Mayo Clinic Foundation

National Institutes of Health/National Institute on Aging

National Institutes of Health/National Institute of Neurological Disorders and Stroke

Amyotrophic Lateral Sclerosis Association

Canadian Institutes of Health Research

Siragusa Foundation

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference20 articles.

1. Modeling key pathological features of frontotemporal dementia with C9ORF72 repeat expansion in iPSC-derived human neurons;Almeida;Acta Neuropathol.,2013

2. Unconventional translation of C9ORF72 GGGGCC expansion generates insoluble polypeptides specific to c9FTD/ALS;Ash;Neuron,2013

3. RNA-mediated toxicity in neurodegenerative disease;Belzil;Mol. Cell. Neurosci.,2012

4. Reduced C9orf72 gene expression in c9FTD/ALS is caused by histone trimethylation, an epigenetic event detectable in blood;Belzil;Acta Neuropathol.,2013

5. Reduced C9orf72 gene expression in c9FTD/ALS is caused by histone trimethylation, an epigenetic event detectable in blood;Belzil;Acta Neuropathol.,2013

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