Inhibition of gp91phox contributes towards normobaric hyperoxia afforded neuroprotection in focal cerebral ischemia-Reference-Cited by-同舟云学术

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Inhibition of gp91phox contributes towards normobaric hyperoxia afforded neuroprotection in focal cerebral ischemia

Published:2010-08 Issue: Volume:1348 Page:174-180
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Tang Xiangqi,Liu Ke Jian,Ramu Jaivijay,Chen Qingquan,Li Ting,Liu Wenlan

Funder

National Institutes of Health

American Heart Association

Foundation of the Ministry of Education of China

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference37 articles.

1. Expression and modulation of an NADPH oxidase in mammalian astrocytes;Abramov;J. Neurosci.,2005

2. Differential matrix metalloproteinase expression in cases of multiple sclerosis and stroke;Anthony;Neuropathol. Appl. Neurobiol.,1997

3. NADPH oxidase;Babior;Curr. Opin. Immunol.,2004

4. Beta-amyloid activates the O-2 forming NADPH oxidase in microglia, monocytes, and neutrophils. A possible inflammatory mechanism of neuronal damage in Alzheimer's disease;Bianca;J. Biol. Chem.,1999

5. Recovery of the cortical evoked response following temporary middle cerebral artery occlusion in baboons: relation to local blood flow and PO2;Branston;Stroke,1976

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