Using hiPSCs to model neuropsychiatric copy number variations (CNVs) has potential to reveal underlying disease mechanisms-Reference-Cited by-同舟云学术

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Using hiPSCs to model neuropsychiatric copy number variations (CNVs) has potential to reveal underlying disease mechanisms

Published:2017-01 Issue: Volume:1655 Page:283-293
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Flaherty Erin K.,Brennand Kristen J.

Funder

Brain and Behavior Young Investigator Grant

National Institute of Health (NIH)

New York Stem Cell Foundation.

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference114 articles.

1. 7Q11.23 dosage-dependent dysregulation in human pluripotent stem cells affects transcriptional programs in disease-relevant lineages;Adamo;Nat. Genet.,2014

2. Common polygenic variation and risk for childhood-onset schizophrenia;Ahn;Mol. Psychiatry,2014

3. High rate of disease-related copy number variations in childhood onset schizophrenia;Ahn;Mol. Psychiatry,2014

4. Endophenotypes in schizophrenia: a selective review;Allen;Schizophr. Res.,2009

5. Isogenic pairs of wild type and mutant induced pluripotent stem cell (iPSC) lines from Rett syndrome patients as in vitro disease model;Ananiev;PLoS One,2011

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2. CRISPR/Cas9 Genome Editing Approaches for Psychiatric Research;Brazilian Journal of Psychiatry;2023

3. Chromosomal Microarray Analysis as First-Tier Genetic Test for Schizophrenia;Frontiers in Genetics;2021-10-01

4. METTL1-mediated m7G methylation maintains pluripotency in human stem cells and limits mesoderm differentiation and vascular development;Stem Cell Research & Therapy;2020-07-22

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