HIF1A overexpression caused by etomidate activates PGK1-induced oxidative stress in postoperative cognitive dysfunction-Reference-Cited by-同舟云学术

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HIF1A overexpression caused by etomidate activates PGK1-induced oxidative stress in postoperative cognitive dysfunction

Published:2024-10 Issue: Volume:1841 Page:149069
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Lu Guangxi,Wen Zhibin,Yu Lu,Wang Chao^ORCID,Gao Yang

Publisher

Elsevier BV

Reference31 articles.

1. Arias-Cavieres, A., et al., 2020. A HIF1a-Dependent Pro-Oxidant State Disrupts Synaptic Plasticity and Impairs Spatial Memory in Response to Intermittent Hypoxia. eNeuro. 7.

2. Expression of miR-200c corresponds with increased reactive oxygen species and hypoxia markers after transient focal ischemia in mice;Arvola;Neurochem Int.,2021

3. Methodology of measuring postoperative cognitive dysfunction: a systematic review;Borchers;Br J Anaesth.,2021

4. Anesthetic agent etiomidate induces apoptosis in N2a brain tumor cell line;Chen;Mol Med Rep.,2018

5. Exploring the modulation of hypoxia-inducible factor (HIF)-1alpha by volatile anesthetics as a possible mechanism underlying volatile anesthetic-induced CNS injury;Giles;Neurochem Res.,2014

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