Mitochondrially targeted vitamin E and vitamin E mitigate ethanol-mediated effects on cerebellar granule cell antioxidant defense systems-Reference-Cited by-同舟云学术

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Mitochondrially targeted vitamin E and vitamin E mitigate ethanol-mediated effects on cerebellar granule cell antioxidant defense systems

Published:2005-08 Issue:2 Volume:1052 Page:202-211
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Siler-Marsiglio Kendra I.,Pan Qun,Paiva Michael,Madorsky Irina,Khurana Nila C.,Heaton Marieta B.

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference43 articles.

1. Ethanol promotes apoptosis in cerebellar granule cells by inhibiting the trophic effect of NMDA;Bhave;J. Neurochem.,1997

2. FGF-2, NGF and IGF-1, but not BDNF, utilize a nitric oxide pathway to signal neurotrophic and neuroprotective effects against alcohol toxicity in cerebellar granule cell cultures;Bonthius;Brain Res. Dev. Brain Res.,2003

3. Ethanol differentially inhibits homoquinolinic acid- and NMDA-induced neurotoxicity in primary cultures of cerebellar granule cells;Cebere;Neurochem. Res.,2003

4. Rapid colorimetric assay for cell growth and survival. Modifications to the tetrazolium dye procedure giving improved sensitivity and reliability;Denizot;J. Immunol. Methods,1986

5. Supplementation of endothelial cells with mitochondria-targeted antioxidants inhibit peroxide-induced mitochondrial iron uptake, oxidative damage, and apoptosis;Dhanasekaran;J. Biol. Chem.,2004

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