Late onset Tay–Sachs disease in mice with targeted disruption of the Hexa gene: behavioral changes and pathology of the central nervous system-Reference-Cited by-同舟云学术

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Late onset Tay–Sachs disease in mice with targeted disruption of the Hexa gene: behavioral changes and pathology of the central nervous system

Published:2004-03 Issue:1-2 Volume:1001 Page:37-50
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Miklyaeva Elena I,Dong Weijia,Bureau Alexandre,Fattahie Roya,Xu Yongqin,Su Meng,Fick Gordon H,Huang Jing-Qi,Igdoura Suleiman,Hanai Nobuo,Gravel Roy A

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference37 articles.

1. Catabolism of asialo-GM2 in man and mouse. Specificity of human/mouse chimeric GM2 activator proteins;Bertoni;J. Biol. Chem.,1999

2. Motor function and the corticospinal tracts in the dog and raccoon;Buxton;J. Comp. Neurol.,1967

3. Gait analysis in the mouse;Clarke;Physiol. Behav.,1999

4. Behaviour, neurology and electrophysiology;Classen,2000

5. Disruption of murine hexa gene leads to enzymatic deficiency and to neuronal lysosomal storage, similar to that observed in Tay–Sachs disease;Cohen-Tannoudji;Mamm. Genome,1995

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1. Treating late-onset Tay Sachs disease: Brain delivery with a dual trojan horse protein;Molecular Therapy - Methods & Clinical Development;2024-09

2. Quantitative brain morphometry identifies cerebellar, cortical, and subcortical gray and white matter atrophy in late‐onset Tay‐Sachs disease;Journal of Inherited Metabolic Disease;2023-12-19

3. The GM2 gangliosidoses: Unlocking the mysteries of pathogenesis and treatment;Neuroscience Letters;2021-11

4. Animal Model Contributions to Congenital Metabolic Disease;Advances in Experimental Medicine and Biology;2020

5. Murine Sialidase Neu3 facilitates GM2 degradation and bypass in mouse model of Tay-Sachs disease;Experimental Neurology;2018-01

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